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Blunt renal trauma-induced hypertension: prevalence, presentation, and outcome.

Created: 14/2/2007
Updated: 15/2/2007


Blunt renal trauma-induced hypertension: prevalence, presentation, and outcome.
Chedid A, Le Coz S, Rossignol P, Bobrie G, Herpin D, Plouin PF.
Am J Hypertens. 2006 May;19(5):500-4.


  • Blunt renal trauma (RT) may cause hypertension.
  • We assessed the frequency and mechanisms of RT, and blood pressure (BP) outcome after treatment


  • We searched the records of all patients referred to our hypertension unit and included those of previously normotensive patients who developed hypertension within 6 months of RT.


  • Ten of the 17,410 referred patients, with a median age of 26 years, developed hypertension 0 to 3 months after a well-documented RT.
  • Median BP at referral was 170/107 mm Hg. Median glomerular filtration rate was 89 mL/min.
  • Five patients had hematuria. Median kidney length was 107 mm on the damaged side and 114 mm on the opposite side.
  • Renal artery lesions were present in six cases. A pattern of unilateral renin hypersecretion and contralateral suppression was present in five of eight cases with unilateral RT.
  • Six patients underwent surgery.
  • Seven months after referral, median BP was 128/79 mm Hg. The BP was <140/90 mm Hg without medication in one patient who did not undergo surgery and in three patients who did.


  • Renal trauma is a rare cause of hypertension, mostly in young men. Hypertension is usually renin dependent and associated with parenchymal injury. The RT-induced hypertension may resolve spontaneously and is amenable to surgery.

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